RELATIONSHIP BETWEEN GENES, PHYSICAL ACTIVITY & METABOLIC DISEASES

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Date
2020-12-15Author
Cerit, Mesut
Dalip, Metin
Yildirim, Damla Selin
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Metabolic diseases appea commonly in all countries of the world with advancing age. Approximately
one in ten people around the world have diabetes (Type II) and others have a risk of diabetes. It is a known
fact that regular physical activity, not only provides a healthy and quality life, it also reduces the risk of
getting chronic diseases such as metabolic and cardiovascular diseases. Many studies have been conducted
in recent years, suggesting that being active or passive can also be caused by genetic factors with a
comprehensive literature base that considers the effects of environmental factors and lifestyle on physical
activity levels. Scientists are trying to reveal the effects of the fatty acid binding protein (fatty acid binding
protein, FABP)-4, which controls the intracellular fatty rate and triggers the shift of metabolism from
balance to imbalance as a result of overnutrition, and (nuclear respiratory factor, NRF)-1 gene, which
ensures the preservation of intracellular harmony. The only alternative method that triggers the transition
from imbalance to stability (stable) is to increase the activity level and make exercise a part of life. Some
genetic structures can arise with many genes and many factors. Genetic infrastructure is susceptible to risk
and this can be observed at a high rate in some people. Physical activities are the most effective application
and protection method that allows the effects of NRF1 gene to increase by reducing the effect of the FABP4
gene. Regular exercises reduce the risk of getting metabolic and chronic diseases such as cancer, blood
pressure and diabetes triggered by genetics and lifestyle by affecting the condition of genetic codes in the
body.
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